WHAT ABOUT STRETCH MARKS:
Stretch Marks (Striae Distensae) and cutaneous lesions that accompany the hormonal upheavals of the major stages of life; Puberty and pregnancy are the main causes. Today, other additional triggering factors may be involved, such as Corticosteroids treatments and obesity. Also, more recently and more unexpectedly, Aesthetic surgery, skin stretching due to breast and buttocks augmentation. Therefore, over 60% of women are affected by stretch marks.
Men are not spared, although, the prevalence is low: puberty, obesity, and intensive body building ( especially, the shoulders, torso and arms), are triggering factors. Stretch marks develop abruptly and progresses slowly from a pink to violet colour , then to an ivory white or sometimes, nacreous hue. They don’t go away oh. They are experienced as unwanted visitors that never leaves.
Although, the exact description of stretch marks have yet been fully understood, there are four determining facts;
- High use or abuse of corticosteroids:. All the findings indicate a strong correlation between endogenous Glucocorticoid secretion and the emergence of stretch marks . The activities of corticosteroids causes the growth of both Keratinocytes and Fibroblasts to As is their synthesis of Collagens 1 and 111, 90% less than normal skin. In addition, Fibronectin is synthesis is reduced; 93% less than normal skin.
- The Early Inflammatory Stage:
A study, including young persons during puberty, presenting with incipient stretch marks showed that stretch marks biopsy specimen were the site of inflammation displaying Granulocyte-Macrophage colony stimulating factor (CSF) and Monocyte – Chemoattractant Protein (MCP). This is accompanied by visible Elastolysis triggered by Mast cell degranulation with release of Trypsin and Chymotrypsin.
What cosmetics treatment of these tissues alterations can do are;
. Prevention of appearance and Propagation on the one hand,
. Repair of some of the tissues damage on the other hand.
They are based on limiting the effect of Mast cell released Trypsin and Chemotrypsin and Macrophage induced Elastolysis by Elastase as well as helping Fibroblast to repair the matrix.
These are the basic functional pathways of EDEN ANTI-STRETCH MARKS